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25 articles
24 juin
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00Potassium-Alkali–Enriched Diet, Hypertension, and Proteinuria following Uninephrectomy - J Am Soc NephrolBackground: Losing or donating a kidney is associated with risks of developing hypertension and albuminuria. Few studies address mechanisms or interventions. We investigate potential benefits of a K+- alkali–enriched diet and the mechanisms underlying proteinuria. Methods: Male Sprague Dawley (…) -- Original Article: Acid Base and Electrolyte Disorders
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07:00Angiotensin-II-Type-1a Receptor (AT1aR) and Renal K+ Wasting during Overnight Low-Na+ Intake - J Am Soc NephrolBackground: Chronic Angiotensin-II (Ang-II) perfusion stimulates Kir4.1/Kir5.1 of the DCT via angiotensin-II-type-1a-receptor (AT1aR) and low-sodium-intake also stimulates Kir4.1/Kir5.1. However, it is not explored the role of AT1aR in mediating the effect of LS on Kir4.1/Kir5.1. Methods: We (…) -- Original Article: Acid Base and Electrolyte Disorders
18 juin
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00Evolutionary Differences in Uric Acid Metabolism Inform about the Risks of Pig-to-Human Kidney Transplant - J Am Soc NephrolNo abstract available -- Perspective
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07:00Association between Acute Declines in Estimated Glomerular Filtration Rate during Renin-Angiotensin System Inhibition and Risk of Adverse Outcomes - J Am Soc NephrolBackground: Declines in glomerular filtration rate (GFR) occur commonly when renin-angiotensin system (RAS) inhibitors are started. Our objective was to determine the relation between declines in estimated GFR during trials of RAS inhibition and kidney outcomes. Methods: We included participants (…) -- Original Article: Chronic Kidney Disease
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07:00Estimated Effect of Restarting Renin-Angiotensin System Inhibitors after Discontinuation on Kidney Outcomes and Mortality - J Am Soc NephrolBackground: While renin-angiotensin system inhibitors (RASi) have been the mainstream treatment for patients with CKD, they are often discontinued due to adverse effects such as hyperkalemia and acute kidney injury. It is unknown whether restarting RASi after discontinuation improves clinical (…) -- Original Article: Chronic Kidney Disease
14 juin
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00
No abstract available -- Editorial
12 juin
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00
Background Information on metabolomic profiles in kidney stone formers is limited. To examine independent associations between plasma metabolomic profiles and the risk of incident, symptomatic kidney stones in adults, we conducted prospective nested case-control studies in two large cohorts. (…) -- Original Article: Acid Base and Electrolyte Disorders
10 juin
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00No Time for Cancel Culture: The Importance of Banff Pathology Criteria and Clinical Outcomes - J Am Soc NephrolNo abstract available -- Editorial
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07:00METTL3-Mediated N6-methyladenosine mRNA Modification and cGAS-STING Pathway Activity in Kidney Fibrosis - J Am Soc NephrolBackground Chemical modifications on RNA profoundly impact RNA function and regulation. m6A, the most abundant RNA modification in eukaryotes, plays a pivotal role in diverse cellular processes and disease mechanisms. However, its importance is understudied in human chronic kidney disease (CKD) (…) -- Original Article: Genetics
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07:00
Background: Low total kidney volume (TKV) is a risk factor for chronic kidney disease (CKD). However, evaluations of nonlinear relationships, incident events, causal inference, and prognostic utility beyond traditional biomarkers are lacking. Methods: TKV, height-adjusted TKV, and body surface (…) -- Original Article: Chronic Kidney Disease
7 juin
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00
Key Points Polycystic kidney disease (PKD) is characterized by continuous cyst growth, which results in a decline in kidney function.Deletion of P2Y2R and pharmacological antagonism of purinergic signaling significantly reduced cyst growth in an orthologous PKD mouse model.P2Y2R was expressed in (…) -- Basic Research: Cystic Kidney Disease
6 juin
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00
No abstract available -- Research Letter
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07:00Glomerular Endothelial Cell Receptor ADGRF5 and the Integrity of the Glomerular Filtration Barrier - J Am Soc NephrolBackground: Glomerular endothelial cells are recognized to be important for maintaining the glomerular filtration barrier. ADGRF5, an adhesion G protein-coupled receptor, has been suggested to be involved in endothelial cell function. However, the role of ADGRF5 in the glomerular filtration (…) -- Original Article: Normal Kidney Structure and Function
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07:00
No abstract available -- Letter to the Editor
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07:00
Background Metabolites represent a read-out of cellular processes underlying states of health and disease. Methods We evaluated cross-sectional and longitudinal associations between 1255 serum and 1398 urine known and unknown (denoted with “X” in name) metabolites (Metabolon HD4, 721 detected in (…) -- Original Article: Chronic Kidney Disease
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07:00Authors' Reply: Utility of Urinary CD4+ T-Cell Count in Detecting ANCA-Associated Vasculitis Renal Relapse - J Am Soc NephrolNo abstract available -- Letter to the Editor
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07:00Utility of Urinary CD4+ T-Cell Count in Detecting ANCA-Associated Vasculitis Renal Relapse - J Am Soc NephrolNo abstract available -- Letter to the Editor
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07:00
No abstract available -- Letter to the Editor
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07:00
No abstract available -- Letter to the Editor
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07:00Authors' Reply: Dialysate Sodium and Mortality: 140 mmol/L at Hemodialysis Initiation by Default? - J Am Soc NephrolNo abstract available -- Letter to the Editor
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07:00Dialysate Sodium and Mortality: 140 mmol/L at Hemodialysis Initiation by Default? - J Am Soc NephrolNo abstract available -- Letter to the Editor
3 juin
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00
No abstract available -- Editorial
31 mai
J Am Soc Nephrol https://jasn.asnjournals.org/
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07:00
No abstract available -- Editorial
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07:00
Background: Secondary hyperparathyroidism of CKD contributes significantly to patient morbidity and mortality. The underlining mechanisms of CKD-induced secondary hyperparathyroidism remain elusive. We previously demonstrated that PT-Dicer-/- mice, with parathyroid specific deletion of the miRNA (…) -- Original Article: Chronic Kidney Disease
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07:00
Background Elderly individuals and chronic kidney disease (CKD) patients are at a higher risk of acute kidney injury (AKI). The transcription factor MondoA is downregulated in the kidneys of aged or AKI patients; however, its roles in AKI development and the AKI-to-CKD transition remain unknown. (…) -- Original Article: Acute Kidney Injury and ICU Nephrology